CHAINE RESPIRATOIRE MITOCHONDRIALE PDF

  • July 24, 2019

paris 12 ue1 biochimie fiche de cours n°13 seances medisup (pr. loric) chaine respiratoire mitochondriale “*les notions indiquées tombent fréquemment au. Les mitochondries sont preparees par centrifugation diffe- rentielle dans un milieu structure de l’antibiotique-activite sur la chaine respiratoire mitochondriale. Le dinitrophénol (DNP) est un composé chimique aromatique de formule C6H4N 2O5. C’est un Le dinitrophénol est un découplant de la chaîne respiratoire mitochondriale. Les intoxications aux dérivés nitrés du phénol sont reconnues.

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Services Articles citing this article CrossRef 6. Write a review Rate this item: Front Endocrinol Lausanne ; 2: Allow this favorite library to be seen by others Keep this favorite library private. Find a copy in the library Finding libraries that hold this item A chwine of brown adipocyte progenitors in human skeletal muscle. In parallel, the recent discovery of significant amounts of BAT in human adults has renewed the interest of the scientific community for this tissue.

Reviews User-contributed reviews Add a review and share your thoughts with other readers. Archives of neurology, 65, 1, p. Beige adipocytes are a distinct type of thermogenic fat cell in mouse and human.

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Linked Data More info about Linked Data. Physiol Rev ; Comparison of the respective influences of the nuclear and mitochondrial T3 pathways demonstrates either both additivity myoblast differentiationcomplementarity mitochondriogenesis, myoblast differentiation or opposite influences myosin expressionthus indicating that these two pathways introduce a fine-tuning of the hormone influence.

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Would you also like to submit a review for this item? The E-mail Address es field is required. PTEN loss in the Myf5 lineage redistributes body fat and reveals subsets of white adipocytes that arise from Myf5 precursors.

NADH déshydrogénase

Muscle physiology in healthy men and women and those with metabolic myopathies Devries MC, Tarnopolsky MA Neurologic clinics, 26, 1, p. Please enter the message. Obes Rev ; In particular, in myoblasts, p43 overexpression stimulates terminal differentiation and induces a preferential expression of slow respirahoire, by down-regulating c-Myc expression and up-regulating calcineurin and myogenin expression.

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Voir aussi Maladie de Charcot-Marie-Tooth de type 4: Current usage metrics About article metrics Return to article. You may have already requested this item. Mitochondrion, 8, 2, p.

P43 activation by T3 stimulates mitochondrial protein synthesis, respiratory chain activity and mitochondriogenesis. Preview this item Preview this item. Diabetes ; Front Endocrinol Lausanne ; 3: Maladie de Charcot-Marie-Tooth de type 4: Pour les lecteurs Alerte courriel.

NADH déshydrogénase — Wikipédia

Stem Cell Rev ; 8: Two mitochondrial import sequences have been characterized in the C-terminal part of these proteins; in addition, their N-terminal part, devoid of negative charges, plays a permissive role in this import. J Cell Sci ; Whereas the function of p28 remains unknown, p43 is a T3-dependent transcription factor of the mitochondrial genome, acting through dimeric complexes involving at least two other truncated forms of nuclear receptors, mtRXR and mtPPAR.

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Advanced Search Find a Library. Given its considerable capacity to dissipate substrates, BAT appears again as a therapeutic target against metabolic diseases such as diabetes and obesity.

Med Sci Paris ; Cell metabolism, 8, 3, p. Journal of physiology The,23, p. Brown adipose tissue-specific insulin receptor knockout shows diabetic phenotype without insulin resistance.

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Sequential changes in the expression of mitochondrial protein mRNA during the development of brown adipose tissue in bovine and ovine species. J Clin Invest ; Occurrence of brown adipocytes in rat white adipose tissue: American journal of human genetics, 95, 3, mjtochondriale. Brown adipose tissue activity controls triglyceride clearance. Search WorldCat Find items in libraries near you.